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dc.creatorKotur-Stevuljević, Jelena
dc.creatorSimić-Ogrizović, Sanja
dc.creatorDopsaj, Violeta
dc.creatorStefanović, Aleksandra
dc.creatorVujović, Ana
dc.creatorIvanić-Corlomanović, Tatjana
dc.creatorSpasić, Slavica
dc.creatorSpasojević-Kalimanovska, Vesna
dc.creatorJelić-Ivanović, Zorana
dc.date.accessioned2019-09-02T11:27:04Z
dc.date.available2019-09-02T11:27:04Z
dc.date.issued2012
dc.identifier.issn0009-9120
dc.identifier.urihttp://farfar.pharmacy.bg.ac.rs/handle/123456789/1623
dc.description.abstractBackground: Atherosclerosis is the main cause of mortality in end stage renal disease (ESRD) patients. Design and methods: Malnutrition, inflammation and diminished paraoxonase activity were used to calculate the sum of risk factors for atherosclerosis development in a cohort of 141 chronic renal disease patients. Kaplan-Meier survival analysis was implemented to assess risk of death. Results: Kaplan-Meier analysis (Log rank = 12.06, P = 0.0072) showed higher risk of death with increasing number of risk factors in haemodialysis patients. Conclusions: Malnutrition in combination with inflammation and oxidative stress is associated with higher mortality in patients on long-term haemodialysis.en
dc.publisherPergamon-Elsevier Science Ltd, Oxford
dc.relationinfo:eu-repo/grantAgreement/MESTD/Basic Research (BR or ON)/175035/RS//
dc.rightsrestrictedAccess
dc.sourceClinical Biochemistry
dc.subjectMalnutritionen
dc.subjectInflammationen
dc.subjectOxidative stressen
dc.subjectParaoxonase-1en
dc.titleA hazardous link between malnutrition, inflammation and oxidative stress in renal patientsen
dc.typearticle
dc.rights.licenseARR
dcterms.abstractСпасојевић-Калимановска, Весна; Стефановић, Aлександра; Јелић-Ивановић, Зорана; Котур-Стевуљевић, Јелена; Допсај, Виолета; Симић-Огризовић, Сања; Иванић-Цорломановић, Татјана; Вујовић, Aна; Спасић, Славица;
dc.citation.volume45
dc.citation.issue15
dc.citation.spage1202
dc.citation.epage1205
dc.citation.other45(15): 1202-1205
dc.citation.rankM21
dc.identifier.wos000309631000016
dc.identifier.doi10.1016/j.clinbiochem.2012.04.021
dc.identifier.pmid22580395
dc.identifier.scopus2-s2.0-84866305373
dc.identifier.rcubconv_2704
dc.type.versionpublishedVersion


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